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Please use this identifier to cite or link to this item: http://acervodigital.unesp.br/handle/11449/36273
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dc.contributor.authorFaine, L. A.-
dc.contributor.authorDiniz, Y. S.-
dc.contributor.authorAlmeida, J. A.-
dc.contributor.authorNovelli, ELB-
dc.contributor.authorRibas, B. O.-
dc.date.accessioned2014-05-20T15:25:58Z-
dc.date.accessioned2016-10-25T18:00:34Z-
dc.date.available2014-05-20T15:25:58Z-
dc.date.available2016-10-25T18:00:34Z-
dc.date.issued2002-05-01-
dc.identifierhttp://dx.doi.org/10.1016/S0278-6915(02)00004-2-
dc.identifier.citationFood and Chemical Toxicology. Oxford: Pergamon-Elsevier B.V., v. 40, n. 5, p. 663-668, 2002.-
dc.identifier.issn0278-6915-
dc.identifier.urihttp://hdl.handle.net/11449/36273-
dc.identifier.urihttp://acervodigital.unesp.br/handle/11449/36273-
dc.description.abstractThe aim of the present study was to determine the effects of ad lib, overfeeding and of dietary restriction (DR) on oxidative stress in cardiac tissue. Lipoperoxide concentrations ere decreased and antioxidant enzymes ere increased in moderate-DR-fed rats. Severe-DR induced increased lipoperoxide concentrations. Overfeeding increased lipoperoxide levels in cardiac tissue. Total superoxide dismutase (SOD) and Cu-Zn superoxide dismutase (Cu-Zn SOD) activities were decreased in cardiac tissue at 35 days of overfeeding. As no changes in glutathione peroxidase (GSH-Px) ere observed in overfed rats, awhile SOD and Cu-Zn SOD activities were decreased in these animals. it is assumed that superoxide anion is an important intermediate in the toxicity of ad lib, overfeeding. Overfeeding induced alterations in markers of oxidative stress in cardiac tissue. (C) 2002 Elsevier B.V. Ltd. All rights reserved.en
dc.format.extent663-668-
dc.language.isoeng-
dc.publisherElsevier B.V.-
dc.sourceWeb of Science-
dc.subjectratpt
dc.subjectreactive oxygen speciespt
dc.subjectoverfeedingpt
dc.subjectdietary restrictionpt
dc.subjectheartpt
dc.titleToxicity of ad lib. overfeeding: effects on cardiac tissueen
dc.typeoutro-
dc.contributor.institutionUniversidade Estadual Paulista (UNESP)-
dc.contributor.institutionInst Salud Carlos III-
dc.description.affiliationUNESP, Inst Biociencias, Dept Chem & Biochem, São Paulo, Brazil-
dc.description.affiliationUNESP, Fac Med, São Paulo, Brazil-
dc.description.affiliationInst Salud Carlos III, Natl Ctr Environm Hlth, Dept Toxicol, Madrid, Spain-
dc.description.affiliationUnespUNESP, Inst Biociencias, Dept Chem & Biochem, São Paulo, Brazil-
dc.description.affiliationUnespUNESP, Fac Med, São Paulo, Brazil-
dc.identifier.doi10.1016/S0278-6915(02)00004-2-
dc.identifier.wosWOS:000175672500011-
dc.rights.accessRightsAcesso restrito-
dc.relation.ispartofFood and Chemical Toxicology-
Appears in Collections:Artigos, TCCs, Teses e Dissertações da Unesp

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