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Please use this identifier to cite or link to this item: http://acervodigital.unesp.br/handle/11449/65080
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dc.contributor.authorDe-Luca, L. A.-
dc.contributor.authorMenani, José Vanderlei-
dc.date.accessioned2014-05-27T11:18:13Z-
dc.date.accessioned2016-10-25T18:14:25Z-
dc.date.available2014-05-27T11:18:13Z-
dc.date.available2016-10-25T18:14:25Z-
dc.date.issued1997-04-01-
dc.identifierhttp://dx.doi.org/10.1590/S0100-879X1997000400011-
dc.identifier.citationBrazilian Journal of Medical and Biological Research, v. 30, n. 4, p. 497-502, 1997.-
dc.identifier.issn0100-879X-
dc.identifier.urihttp://hdl.handle.net/11449/65080-
dc.identifier.urihttp://acervodigital.unesp.br/handle/11449/65080-
dc.description.abstractWater and saline intake is controlled by several mechanisms activated during dehydration. Some mechanisms, such as the production of angiotensin II and unloading of cardiovascular receptors, activate both behaviors, while others, such as the increase in blood osmolality or sodium concentration, activate water, but inhibit saline intake. Aldosterone probably activates only saline intake. Clonidine, anα2-adrenergic agonist, inhibits water and saline intake induced by these mechanisms. One model to describe the interactions between these multiple mechanisms is a wire-block diagram, where the brain circuit that controls each intake is represented by a summing point of its respective inhibiting and activating factors. The α2-adrenoceptors constitute an inhibitory factor common to both summing points.en
dc.format.extent497-502-
dc.language.isoeng-
dc.sourceScopus-
dc.subjectα2-Adrenergic agonist-
dc.subjectClonidine-
dc.subjectDehydration-
dc.subjectNoradrenaline-
dc.subjectSodium intake-
dc.subjectWater intake-
dc.subjectalpha 2 adrenergic receptor-
dc.subjectalpha adrenergic receptor stimulating agent-
dc.subjectclonidine-
dc.subjectnoradrenalin-
dc.subjectsodium chloride-
dc.subjectanimal-
dc.subjectdehydration-
dc.subjectdrinking-
dc.subjectdrug effect-
dc.subjectmetabolism-
dc.subjectphysiology-
dc.subjectrenin angiotensin aldosterone system-
dc.subjectreview-
dc.subjectAdrenergic alpha-Agonists-
dc.subjectAnimals-
dc.subjectDrinking-
dc.subjectNorepinephrine-
dc.subjectReceptors, Adrenergic, alpha-2-
dc.subjectRenin-Angiotensin System-
dc.subjectSodium Chloride-
dc.titleMultifactorial control of water and saline intake: Role of α2-adrenoceptorsen
dc.typeoutro-
dc.contributor.institutionUniversidade Estadual Paulista (UNESP)-
dc.description.affiliationDepto. de Cie. Fisiológicas Faculdade de Odontologia Universidade Estadual Paulista, 14801-903 Araraquara, SP-
dc.description.affiliationUnespDepto. de Cie. Fisiológicas Faculdade de Odontologia Universidade Estadual Paulista, 14801-903 Araraquara, SP-
dc.identifier.doi10.1590/S0100-879X1997000400011-
dc.identifier.scieloS0100-879X1997000400011-
dc.identifier.wosWOS:A1997WT02000011-
dc.rights.accessRightsAcesso aberto-
dc.identifier.file2-s2.0-0031110231.pdf-
dc.relation.ispartofBrazilian Journal of Medical and Biological Research-
dc.identifier.scopus2-s2.0-0031110231-
Appears in Collections:Artigos, TCCs, Teses e Dissertações da Unesp

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