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Please use this identifier to cite or link to this item: http://acervodigital.unesp.br/handle/11449/68755
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dc.contributor.authorVieira, Alexandre A.-
dc.contributor.authorDe Paula, Patricia M.-
dc.contributor.authorDe Luca Jr., Laurival A.-
dc.contributor.authorColombari, Débora S. A.-
dc.contributor.authorColombari, Eduardo-
dc.contributor.authorMenani, José Vanderlei-
dc.date.accessioned2014-05-27T11:21:48Z-
dc.date.accessioned2016-10-25T18:21:53Z-
dc.date.available2014-05-27T11:21:48Z-
dc.date.available2016-10-25T18:21:53Z-
dc.date.issued2006-01-01-
dc.identifierhttp://www.revistas.usp.br/rmrp/article/view/361-
dc.identifier.citationMedicina, v. 39, n. 1, p. 21-27, 2006.-
dc.identifier.issn0076-6046-
dc.identifier.urihttp://hdl.handle.net/11449/68755-
dc.identifier.urihttp://acervodigital.unesp.br/handle/11449/68755-
dc.description.abstractThe maintenance of the arterial pressure in normal levels is important for the homeostasis of body fluids. The central nervous system regulating sympathetic and parasympathetic autonomic efferent can adjust arterial pressure which allows animals or human to face different daily activities with the best performance. Different central areas are responsible for the control of autonomic discharges to cardiovascular system and many of them are also involved in the control of fluid electrolyte balance. One of these areas is the tissue surrounding the anteroventral third ventricle (AV3V region) localized in the forebrain and a main central site for angiotensin II receptors and osmoreceptors. The AV3V lesions impair the development of many models of experimental hypertension in rats and the pressor responses to different stimuli. Lesions of the AV3V region also reduce dipsogenic responses to angiotensin II, central cholinergic activation, water deprivation and increase in plasma osmolarity, atrial natriuretic peptide secretion produced by body fluid expansion and the increase in renal excretion to central cholinergic activation. Recent evidence also suggests the participation of AV3V region in pressor responses produced by the activation of medullary mechanisms.en
dc.format.extent21-27-
dc.language.isopor-
dc.sourceScopus-
dc.subjectAngiotensin-
dc.subjectGlutamate-
dc.subjectHypertension-
dc.subjectHypothalamus-
dc.subjectSympathetic Nervous System-
dc.subjectangiotensin-
dc.subjectangiotensin 2 receptor-
dc.subjectarterial pressure-
dc.subjectautonomic nervous system-
dc.subjectbrain third ventricle-
dc.subjectcardiovascular function-
dc.subjectcardiovascular system-
dc.subjectcentral nervous system-
dc.subjectcholinergic activity-
dc.subjectconference paper-
dc.subjectelectrolyte balance-
dc.subjectexperimental model-
dc.subjectforebrain-
dc.subjecthomeostasis-
dc.subjecthypertension-
dc.subjecthypothalamus-
dc.subjectnonhuman-
dc.subjectosmoreceptor-
dc.subjectplasma osmolarity-
dc.subjectpressor response-
dc.titleImportância da região anteroventral do terceiro ventrículo (AV3V) no controle cardiovascular e do equilíbrio hidroeletrolíticopt
dc.title.alternativeImportance of the anteroventral third ventricle (AV3V) region in cardiovascular control and hydromineral balanceen
dc.typeoutro-
dc.contributor.institutionUniversidade Estadual Paulista (UNESP)-
dc.description.affiliationFaculdade de Odontologia de Araraquara - UNESP Programa de Pós-graduação em Ciências Fisiológicas UFSCar, Araraquara - SP-
dc.description.affiliationFaculdade de Odontologia de Araraquara - UNESP, Araraquara - SP-
dc.description.affiliationDepartamento de Fisiologia e Patologia Faculdade de Odontologia de Araraquara - UNESP, Rua Humaitá, 1680, CEP 14.801 903, Araraquara - SP-
dc.description.affiliationUnespFaculdade de Odontologia de Araraquara - UNESP Programa de Pós-graduação em Ciências Fisiológicas UFSCar, Araraquara - SP-
dc.description.affiliationUnespFaculdade de Odontologia de Araraquara - UNESP, Araraquara - SP-
dc.description.affiliationUnespDepartamento de Fisiologia e Patologia Faculdade de Odontologia de Araraquara - UNESP, Rua Humaitá, 1680, CEP 14.801 903, Araraquara - SP-
dc.rights.accessRightsAcesso aberto-
dc.identifier.file2-s2.0-33750282663.pdf-
dc.relation.ispartofMedicina-
dc.identifier.scopus2-s2.0-33750282663-
Appears in Collections:Artigos, TCCs, Teses e Dissertações da Unesp

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